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TheRoleofCirculatingBiomarkersinPost-COVID-19 Patients

The new coronavirus infection has made significant adjustments to the condition of patients already suffering from cardiovascular diseases. Similar mechanisms of fibrosis in patients with severe COVID-19 and heart failure have drawn attention to inflammatory biomarkers. The severe cytokine storm that is characteristic of COVID-19 is critical to understanding the outcome of the disease. In severe COVID-19, higher levels of interleukin-2, interleukin-6, interleukin-10, monocyte chemotactic protein-1 (MCP-1), and tumor necrosis factor-α (TNF-α) were noted than in patients with mild to moderate form of the disease. Elevated levels of MCP-1 in severe COVID-19 suggest that its secretion may play a key role in the reactive inflammatory process associated with COVID-19. It has been established that IL-10 and MCP-1 are predictors of the severity of COVID-19 disease and the risk of death in such patients. Matrix metalloproteinases (MMPs) are also involved in the development of lung pathology and may act as an early indicator of respiratory failure in patients with COVID-19. In this review, biomarkers of inflammation, such as MMP, tissue inhibitor of matrix metalloproteinase-1, interleukins, MCP-1, are considered for their ability to serve as objective markers for early diagnosis and prognosis of the development of cardiovascular complications in patients who have undergone COVID-19.

DOI: 10.52575/2687-0940-2023-46-3-231-244
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